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Mechanisms of Secondary Brain Damage from Trauma and Ischemia Recent Advances of
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- Compralo SubitoMechanisms of Secondary Brain Damage from Trauma and Ischemia Recent Advances of
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Oggetto che si trova a: Livonia, Michigan, Stati Uniti
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Consegna prevista tra il mar 5 ago e il mar 12 ago a 94104
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Numero oggetto eBay:156488408966
Specifiche dell'oggetto
- Condizione
- Product Type
- Textbook
- Topic
- Medical
- Educational Level
- Professional
- ISBN
- 9783211209325
Informazioni su questo prodotto
Product Identifiers
Publisher
Springer
ISBN-10
3211209328
ISBN-13
9783211209325
eBay Product ID (ePID)
30781109
Product Key Features
Number of Pages
VIII, 132 Pages
Publication Name
Mechanisms of Secondary Brain Damage from Trauma and Ischemia : Recent Advances of Our Understanding
Language
English
Publication Year
2004
Subject
Neurology, Emergency Medicine, Surgery / Neurosurgery, Critical Care
Type
Textbook
Subject Area
Medical
Series
Acta Neurochirurgica Supplement Ser.
Format
Hardcover
Dimensions
Item Weight
22 Oz
Item Length
11 in
Item Width
8.3 in
Additional Product Features
Intended Audience
Scholarly & Professional
LCCN
2004-048154
Dewey Edition
22
Series Volume Number
89
Number of Volumes
1 vol.
Illustrated
Yes
Dewey Decimal
617.4/81044
Table Of Content
Secondary brain damage -- the basic sciences.- Knowledge matters.- Gene profiling -- chances and challenges.- Role of mitochondrial proteins for neuronal cell death after focal cerebral ischemia.- Signalling mechanisms for survival of lesioned motoneurons.- Genetically modified animals in molecular stroke research.- Progress in the understanding of brain injury from ischemia.- The Janus face of inflammation in ischemic brain injury.- The natural course of lesion development in brain ischemia.- Pharmalogical preconditioning in global cerebral ischemia.- Traumatic brain and spinal cordinjury -- the experimental approach.- The role of inflammatory processes in the pathophysiology and treatment of brain and spinal cord trauma.- L-type calcium channel antagonist nifedipine reduces neurofilament restitution following traumatic optic nerve injury.- Neuronal activity and cortical perfusion determined by quantitative EEG analysis and laser Doppler flowmetry are uncoupled in brain injured rats.- Glial scar and axonal regeneration in the CNS: lessons from GFAP and vimentin transgenic mice.- Traumatic brain and spinal cord injury -- clinical progress including treatment.- Recovery from spinal cord injury -- underlying mechanisms and efficacy of rehabilitation.- Lessons from epidemiologic studies in clinical trials of traumatic brain injury.- The MRC CRASH trial -- a large, simple randomised trial of steroids in head injury.- Clinical trials in traumatic brain injury: current problems and future solutions.- Prospective documentation and analysis of the pre- and early clinical management in severe head injury in Southern Bavaria at a population based level.- Author Index.- Index of Keywords.
Synopsis
The international interdisciplinary gathering of top of secondary brain damage in brain trauma, as ac level clinical and laboratory scientists in Mauls, Italy knowledged from the beginning of these workshops in has developed from its beginning in 1984into a fruitful Mauls, the significance of inflammation is all but clear. tradition where worldwide experts active and knowl Although inflammatory phenomena are seen in trauma edgeable in cerebral ischemia and trauma convene for and ischemia ofthe brain, as activation ofwhite blood update and exchanges of their most recent clinical and cells with emigration into the tissue presumably en experimental findings and concepts. These meetings hancing damage, inflammatory cells may have benefi have, of course, experienced shifts in emphasis from cial properties as well. Thechapter on the Janus-faceof the past until now, corresponding to the most actual inflammation isanalyzing this ambiguity. developments, which were fascinating clinicians and The exploration of novel cell-biological mechanisms laboratory scientists alike. The current Supplement of on a molecular or more systemic basis causing apop Acta Neurochirurgica is an example in case. Its virtue totic cell death, inflammation, or regeneration, provide as before is that authors contribute articles in a review useful objectives for therapeutical interventions ex like manner on their own field of research, according pected to be more specific than the present treatment to the platform presentations at the meeting as indis modalities.", The international interdisciplinary gathering of top of secondary brain damage in brain trauma, as ac level clinical and laboratory scientists in Mauls, Italy knowledged from the beginning of these workshops in has developed from its beginning in 1984into a fruitful Mauls, the significance of inflammation is all but clear. tradition where worldwide experts active and knowl Although inflammatory phenomena are seen in trauma edgeable in cerebral ischemia and trauma convene for and ischemia ofthe brain, as activation ofwhite blood update and exchanges of their most recent clinical and cells with emigration into the tissue presumably en experimental findings and concepts. These meetings hancing damage, inflammatory cells may have benefi have, of course, experienced shifts in emphasis from cial properties as well. Thechapter on the Janus-faceof the past until now, corresponding to the most actual inflammation isanalyzing this ambiguity. developments, which were fascinating clinicians and The exploration of novel cell-biological mechanisms laboratory scientists alike. The current Supplement of on a molecular or more systemic basis causing apop Acta Neurochirurgica is an example in case. Its virtue totic cell death, inflammation, or regeneration, provide as before is that authors contribute articles in a review useful objectives for therapeutical interventions ex like manner on their own field of research, according pected to be more specific than the present treatment to the platform presentations at the meeting as indis modalities.
LC Classification Number
RD592.5-596
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